What are they, and why should you know about this important element of the aging process?

Well telomeres in fact work much like a clock.

It’s sequential.

Chronologically, or as time passes, telomeres degrade with oxidative damage, radiation, DNA errors in replications such as mutations, epigenetics a.k.a. what genes you have inherited from your parents and consequently ancestors. Epigenetics, sequential. A gene was turned on (or off) in the past that codes for a certain trait such as the processes of aging, and ultimately death and self-preservation.

So, what’s the point here.

Well it is simple…

As you age your telomeres shorten.

There is a direct link between telomere length at conception and death with aging itself.

So, keep your telomeres long, strong, and on.


Well antioxidants are key here.

Biasco (2007) I’m sure will back me up here in the same way I back this statement by him:

“Telomere shortening occurs concomitant with organismal aging, and it is accelerated in the context of human diseases associated with mutations in telomerase, such as some cases of dyskeratosis congenita, idiopathic pulmonary fibrosis and aplastic anemia. People with these diseases, as well as Terc-deficient mice, show decreased lifespan coincidental with a premature loss of tissue renewal, which suggests that telomerase is rate-limiting for tissue homeostasis and organismal survival.”

Here’s the plan:

Telomeres, the encapsulatory parts of chromosones that shorten with age where longer ones mean longer lifespans (Amen, 2017).

Learn something new. Everyday. No exceptions.

Sleep. Get some.

Antioxidants are essential. Sugar and stress cause oxidative damage, making them the enemy.

Take Alpha-GPC; associated with improved telomere sustainability.

Be social. Volunteer etc.

That’s it!


Amen, D. (September, 2017. Does your brain have to age. Refer to

Blasco, M. A. (2007). Telomere length, stem cells and agingNature chemical biology3(10), 640.

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